Sentence examples for binding of the complement from inspiring English sources

Because there are no complement regulatory proteins – which usually inhibit complement activation on eukaryotic cells – present on the cartilage surface and because the bound IgGs are able to bind C3b and prevent the binding of the complement regulatory plasma proteins factor I and factor H, formation of the C3 convertase is possible.

Neu5Ac is a negatively charged 9-cabon sugar that inhibits complement, in part by enhancing binding of the complement inhibitor factor H (FH) through C-terminal domains (19 and 20) on FH.

The immunoglobulin superfamily is a family of cell surface receptors, which participate in many tasks, including antigen recognition and binding of the complement.

The aim of the present study was to investigate to what extent CS-A contributes to the binding of the complement recognition molecule C1q and the complement regulators C1 inhibitor (C1INH), C4b-binding protein (C4BP), and factor H to platelets.

Strain FA1090 is inherently resistant to the bactericidal activity of NHS due to the binding of the complement regulatory protein human C4b-binding protein (hC4BP) to its porin [31].

However, the binding of complement proteins is independent of complement activation, and inhibition of complement at the stage of C1q or C3 does not affect the binding of the complement components.

Immunological and biochemical findings [ 24, 27] have further emphasised the importance of paramyosin in protecting schistosomes from immune attack by binding proteins of the complement pathway [ 28].

For example, deletions of alb1 and arp1 significantly increase the binding of the human complement component C3 to A. fumigatus conidia [46], [51].

In contrast, a novel 45 kDa protein was shown to strongly interact with C4b-binding protein suggesting that in B. recurrentis at least two different receptors for binding of the two complement regulators, CFH and C4b-binding protein, are expressed (unpublished).

This suggests that other factors prolong the lifetime of (CTG n and (CAG n hairpins in vivo, among which may be MMR complexes, negative supercoiling behind replication or transcription forks, replication fork reversal, and protein, RNA, or leading strand binding of the hairpin complement.

Of interest, the four strains in the Farm B clade had a premature stop-codon, Ile369Stop in sbi (SARLGA251_22000), encoding a protein involved immunoglobulin binding and evasion of the complement system (Haupt et al, 2008; Table 1).