Sentence examples for binding of neurons from inspiring English sources

ADAM15, which codes for a disintegrin metalloprotease, has been implicated in both the process of neuronal hypoxic injury [ 35] and the protection (via GRP78 binding) of neurons from hypoxia-induced apoptosis [ 74].

Gamma rhythm is thought to come from the "binding" of neurons, as cells distributed across the cortex draw themselves together into a new network, which is then able to enter consciousness.

Contribution of multiple binding sites.

In this study, we demonstrated for the first time that A β42O induces the depletion of surface β1-integrin via its direct binding to low-intermediate activation conformers of β1-integrin, as detected by competition of A β42O binding to neurons and HT22 cells with the β1-integrin activating antibody (HUTS-4) or MnCl2, both of which induce high conformational activation of β1-integrin.

Unlike in vitro binding assays, there was no evidence of the chGantenerumab binding to neurons.

Focusing on the downstream effects of Aβ42 oligomer binding to neurons, Susan Catalano's (Cognition Therapeutics, Inc).

The antibody chGantenerumab also bound with high affinity to immobilised Aβ peptide, but showed strong binding to neurons in cryosections of both transgenic and wild-type animals.

Based on previous research, the authors of the study postulated that at the molecular level, painkillers binding to neurons on the periphery of the body cause these two unwanted side effects, while the pain relief occurred at sites in the brain and spinal cord.

MAG binding to neurons results in stabilization of the axon, but inhibits further neurite extension (Pan et al., 2005).

It is noteworthy that infliximab did not block oligomer binding to neurons (Fig 3G), substantiating the notion that activation of TNF-α/eIF2α-P signaling is independent of direct binding of AβOs to individual neurons and is likely mediated by TNF-α secreted to the medium.

There were no significant differences in the amounts of cell-bound Aβ42 between control and squalestatin-treated neurons, indicating that cholesterol depletion did not affect the binding of Aβ42 to neurons.