Sentence examples for binding is supported by from inspiring English sources
Furthermore, a possible ring-opening mechanism (off-on) of the rhodamine spirolactam induced by Cu2+ binding is supported by Job plot, ESI-mass, FT-IR, and 1H NMR.
A role for tissue-specific Ubx binding is supported by a global analysis of the binding sites.
This possibility of such a binding is supported by the observation that the oscillation patterns of LOV1 protein levels and CO mRNA levels were largely in reverse phase to each other (Figures 2 and 3).
The hypothesis of a specific binding is supported by the results of the in vitro experiments, which demonstrated a higher accumulation in the known CAIX-positive SKRC 52 cell line [29], while the binding of the peptide was reduced to the background level for the cell line CaKi 2 which has been described as CAIX negative [29].
The suggestion that MutS T-bulge coMutS T-bulgergo a transition from B to U too quicomplexesobservation undergoitial binding is supportransitiondies ofromse-flipping enzymes that induce DNA Bending prior to DNA Unbending and base flipping.
The lack of an effect of the H216N mutation on nucleotide binding is supported by the similarity of the kinetics of MgFTP binding and dissociation between wild-type and H216N RePC.
These data indicate that the translocation process of ADP-ribosylating toxins through pores formed by oligomeric toxin binding components is supported by a complex intracellular machinery.
To decipher which of these three binding sites is supported by the β-catenin/LEF1 complex, reporter gene assays with constructs harboring sites for either IRF3, AP-1 or NF-кB were performed.
The functional significance of the HLS1 EIN3 binding site is supported by a previous study that identified two allelic mutations in the HLS1 promoter sufficient to yield a 'hookless' phenotype (Lehman et al., 1996).
15) The suggestion in the Discussion that SSB-interacting partners might modulate the condensation state of the SSB-ssDNA fiber by altering the binding mode is supported by work on the interaction between SSB and both PriA and PriC (Bhattacharyya et al. 2014, Wessel et al. 2013).
The idea that H3K79 may act as an anti-binding signal is supported by the observation that binding of Sir3 to chromatin is negatively affected by methylation of H3K79 [ 2, 7, 25, 27, 32- 36], and that the presence of Dot1 delays the re-establishment of Sir3-mediated silencing of a previously derepressed (and presumably H3K79 methylated) locus [ 34, 36].
