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Exact(10)
Repressor function in EcBirA is in association with bio-5'-AMP (co-repressor) and hence very tight binding is required to perform this function (26).
TFIIIC binding is required to recruit TFIIIB at most Pol III promoters.
CTCF binding is required to maintain the maternal IC1 hypomethylated during early embryogenesis (9).
We therefore conclude that RNA-stimulated ATP binding is required to induce signaling by RIG-I.
As in the case of PP5, substrate binding is required to stimulate the activity of DUSP1, DUSP2, DUSP6 and DUSP9.
KrasG12D expression is necessary but not sufficient to initiate neoplasia; GTP binding is required to activate KrasG12D (Huang et al., 2014).
Similar(49)
Furthermore, we found that ATP binding was required to reinitiate processive stepping after the terminal backstep.
The observation of cellular colocalization and physical binding are required to demonstrate a cellular interaction between two components.
Apparently cell surface binding was required to detach the viral DNA from the core membrane and de-condense it, such that it could be released as a whole into the cytoplasm for subsequent cytoplasmic DNA-replication.
To overcome this, the basal rate of Gt binding was required to be significantly faster.
To gain further insights into the inhibition of the NLS function in BCR-ABL, we focused on (a) the inverse correlation between BCR-ABL kinase activity and its nuclear import, and (b) the notion that F-actin-binding is required to retain BCR-ABL in the cytoplasm.
More suggestions(17)
binding is observed to
binding is thought to
binding is seen to
binding is combined to
binding is coupled to
binding is confined to
binding is meant to
binding is assumed to
binding is found to
binding is reported to
binding is shown to
binding is indicated to
binding is transmitted to
binding is suggested to
binding is taken to
binding is limited to
binding is linked to
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