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In contrast, mRNA levels for OP, which favors osteoclast binding is increased by stimulation with TGF-β1 and over-expression of caAlk5.
Irreversible binding is increased by microsomal enzyme induction and by anaerobic conditions.
AUF1-PTH mRNA binding is increased by hypocalcemia or chronic kidney disease, or decreased by hypophosphatemia or administration of the calcimimetic R568 [ 7, 15].
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We showed that Egr-1 DNA binding was increased by TNFα in a U0126-sensitive fashion.
However, NF- κB DNA binding was increased by 50 and 75 μ M NS-398 at 72 and 96 h, when NS-398-induced apoptosis is maximal.
We found that ectopically expressed Ebp1 bound PAK1 in MCF-7 cells and that this binding was increased by HRG treatment.
The quantity of BP binding was increased by prior intratracheal treatment of hamsters with BP plus Fe2O3 in vivo, this induced binding was inhibited by addition of 7,8-benzoflavone to the incubation medium.
In a separate recent study, we demonstrated that the on-rate of ligands to the conformational gp120 co-receptor binding site is increased by conformational stabilization, resulting in an increase of antibodies targeted to the stabilized site [55].
The same technology enabled us to show that the reactivity of 4B7 mAb to NPM-CL complexes was markedly enhanced in comparison with its reactivity to either NPM or CL alone, suggesting that 4B7 is representative of mAbs exhibiting a dual specificity similar to that previously reported for certain anti-histone murine mAbs, whose binding activity is increased by DNA [ 22, 26].
Interaction of budding yeast Stn1 with overhang-binding Cdc13 is increased by Cdc13 SUMOylation.
The Ca2+ concentration is tightly regulated by multiple Ca2+ channels, pumps, and binding proteins; [Ca2+]i is increased by Ca2+ influx across the plasma membrane and Ca2+ release from intracellular stores.
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