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In addition, GH receptor expression and GH binding increase in an estradiol-dependent manner in rat and human bone cell lines [ 32], and this relationship between estradiol and GH extends to the liver [ 33] and ovaries [ 34].
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As shown in Figure 1A, between 10 and 120 nmol/L 3H-FA, the total binding increased in a linear fashion.
When binding of 125I-Cry2Ab was measured at increasing concentrations of BBMV, specific binding increased in the susceptible strain in a dose dependent manner, however, a very strong reduction in binding was observed with BBMV from the resistant strain (Fig. 3A).
In contrast, SP-D binding increased in the DeltacnaA strain only after nikkomycin Z treatment.
In each case, USF1 binding increases in Clock Δ19 mutants.
The strength of binding increased in the order B3VH/B3 R27aS VL
Consistent with this notion, previous reports have shown that FOXP3 DNA binding is increased, in a cyclosporine A-sensitive manner, upon stimulation of T cells through the T cell antigen receptor [25], [30], [40].
In addition, we found that the background levels of Orc2 and Mcm7 binding increase throughout the episomes in ΔDS p2910) suggesting an increased licensing at several auxiliary initiation sites.
Based on MRK 16 binding, an increase in P-gp levels was seen in the CEM/A7R line after a 24 h exposure to 1 microgram ml-1 daunorubicin or epirubicin.
TGFβ treatment resulted in a decrease in c-Jun binding and an increase in JunB binding.
An antibody can recognize its antigen, and can evolve toward the immunogen to increase binding strength, in a process referred to as affinity maturation.
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