Sentence examples for binding in the midbrain from inspiring English sources

During and after exposure, we assessed nicotinic cholinergic receptor binding in the midbrain, cerebral cortex, and hippocampus, using [3H]cytisine.

However, in the present study, SERT binding in the midbrain correlated significantly with DAT binding in the most affected putamen and whole striatum (Figure 2).

In humans, [123I]FP-CIT binding in the midbrain and diencephalon could be blocked by a selective serotonin reuptake inhibitor [22].

The adolescent onset nicotine-self-administering rats also had significantly greater high affinity nicotinic receptor binding in the midbrain and the striatum, whereas hippocampal binding did not differ between the age groups.

Average variability was 10%, which is comparable to previously published variability in extrastriatal [123I]FP-CIT binding in the midbrain [29] and only slightly higher than variability in striatal [123I]FP-CIT binding, which is ~ 7.5%[22]].

After adolescent nicotine treatment (postnatal days 30-47), PXT binding was reduced in the hippocampus and striatum instead of the cerebral cortex, again accompanied by increased binding in the midbrain and brainstem; the patterns of effects within each region were gender-selective, although both males and females displayed abnormalities.

Bipolar patients had slightly higher binding potentials in the midbrain than the healthy controls, while patients with major depression had slightly lower binding potentials than the healthy controls.

There were no significant differences in binding potentials in the midbrain compared to the mood-disorder patients and the healthy controls.

In addition, in an autoradiographic study of the postmortem human brain, [125I]β-CIT binding in the thalamus, hypothalamus, and midbrain, with the exception of the substantia nigra, could be completely displaced by addition of the selective serotonin reuptake inhibitor citalopram, indicating that binding in these areas is almost exclusive to SERT and not to dopamine transporters [28].

In the case of molecular functions, we remark tropomyosin binding and actin binding for heart (because of the heart muscle), antiporter activity for the kidney, receptor binding for the lung, and GTP binding for the midbrain (signal transduction).

In a post hoc subgroup analysis, the purging bulimics had higher SERT binding than the healthy women in the midbrain (p = 0.03), but not in the thalamus.