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Insulin autoantibody binding has been noted not to be normally distributed.
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The absence of correlation of the enzymatic activity of SigA with its binding activity has been noted for another related SPATE autotransporter, Tsh [16].
The similarity of the YrbE and Mce proteins with ATP- binding cassette (ABC) transporter permeases and substrate-binding proteins, respectively, has been noted previously [ 8, 9].
It has been noted that tubulin binding and subsequent dynamic suppression contribute to the benefits but also to the toxic side effects of these drugs [ 2].
The abundance of these putative RNA binding proteins among collections of cloned EMB genes has been noted before [37], [38].
In descriptions of SPC structure, the NAD+-binding domain has been identified as a Rossmann fold, but it has been noted that this assignment is not certain.
Exacerbations has been noted during premenstrual phase.
In the current study, we performed western blot analysis on independent NAF samples to confirm the overexpression of vitamin D-binding protein that had been noted with ICAT.
Three residues (27, 54 and 89) from our predictions (red colored in Figure 9) contribute to substrate-binding specificity, as have been noted before in the structural studies of the UMP kinases [ 38]. Figure 9 highlights our predicted functional residues on the adenylyl kinase protein structure.
Previous nuclear, non-phosphatase-dependent effects of PTEN have been noted, including binding and activation of p53, and binding and inactivation of the nuclear proto-oncogene microspherule protein 58 (MSP58).
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