Sentence examples for binding could be due from inspiring English sources

However, the lack of binding could be due to low assay sensitivity, as noted previously [42].

However, these differences in observed binding could be due to multiple binding sites on the DNA molecules, as opposed to the two known FnIII9-10 binding sites on the Fn dimer.

The PDIM synthesis defect in H37Ra and the loss of neutral red binding could be due to a severe truncation in the nrp gene (non-ribosomal peptide synthetase) and the two mutations in pks12 identified in this study (Table 2, Table S3), which are expected to affect the virulence of H37Ra.

The resulting poor binding could be due to the lesion itself or the nucleotide binding properties of E295K.

This decreased binding could be due to decreased PARP-1 expression, differential levels of PARP-1 activity and/or accessibility of PARP-1.

Non-exclusively, abundant mRNA despite decreased levels of RNAPII binding could be due to a switch from labile to stable transcript states, consistent with previous studies demonstrating a large increase in transcript stability over the course of the IDC [ 12].

Drugs may compete with each other for binding to albumin, which suggests some specificity of binding, but could be due to nonspecific occlusion of a hydrophobic patch available for contact [ 10].

This more restricted binding pattern could be due to the activity of other chromatin-binding modules, including the N-terminal AT hooks of MLL1 which have been demonstrated to bind AT-rich regions of DNA [ 67] and a PHD finger (PHD3) which may recognize specific histone methylation marks [ 68– 71].

The differences in binding behavior could be due to experimental conditions or be inherent in the nature of motor binding to MTs.

The apparent discrepancy between the in vivo and in vitro binding assays could be due to different folding of the protein under the two different conditions, suggesting that the Y15A, K186Q, or LL241 242AA mutations might affect the conformation or the higher-order structure of HIV-1 IN that is required for efficient interaction with SIP1 in vivo.

This binding pose could be due to the introduction of bulk (para-substituted methoxyphenyl groups) at the N4-position of the triazole ring.