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The marked dissimilarity between the fast (nonspecific) and slow (specific) binding components was also consistent with a parallel model.
Similar(59)
IG3 binding to ECM components was also observed pre-coating the wells at lower concentrations (10 μg/ml), showing a significant concentration-dependent binding of IG3 to laminin, fibronectin, and collagen type I (data not shown).
The presence of auxiliary origins in the EBV genome is consistent with the observation that in Raji and other Burkitt's lymphoma cell lines the binding of pre-RC components is also not restricted to oriP [27], [28].
A reaction of the pollutants with the components is also conceivable preventing binding on the active site of the enzyme.
Individual components are also listed.
A national component was also added.
However, for the O 1s spectrum, an additional peak low-intensity higher binding energy component is also observed.
The racial component is also distressing.
The writing component is also problematic.
The binding component is divided into 4 domains.
Silver binding was also investigated.
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