Sentence examples for binding component in the from inspiring English sources

Exact(3)

Furthermore, we observe evidence for a specific α7 nAChR binding component in the large arteries supplying the brain.

Without CaCO3, the remaining C S H, which acts as the binding component in the hydrated cement, is converted into amorphous silica gel (amSiO2), as expressed by Eq. (7).

In line with this, the nonspecific binding component in the 3 nM BODIPY-TMR-CGP dissociation trace obtained in CHO-β1 cells was also too low to be detected and therefore was analyzed as β1-adrenoceptor-specific dissociation using a 1-phase dissociation equation.

Similar(57)

Although YntA (the periplasmic Ni-binding component in the YntABCDE system) is evolutionarily distant from NikA, and it is still unclear how YntA binds Ni, gene neighborhoods could be used to identify this distant Ni ABC-transporterorter family.

Given the impossibility of finding a suitable reference region, data were then normalized to total brain uptake and are illustrated in Figure 3 (bottom row) for kinetic models incorporating the TSPO-binding component in the endothelium (right panel) or not (left panel).

Our current results can neither exclude a retrospective binding component in "action + tone" trials when the action – outcome delay is fixed, nor, indeed, in "action only" trials, as the omission of an expected outcome in these trials may well constitute a sensory event (Sutton, Tueting, Zubin, & John, 1967) and thus alter action awareness retrospectively.

We used the human epidermal growth factor (EGF) as a binding component in an EGF-MAP fusion protein to test the efficacy of MAP.

We previously reported that BSSL is the major DC-SIGN binding component in human milk, but mucin 1 - mainly present in milk lipid vesicles - also binds to DC-SIGN [25].

The other two components of the ABC superfamily transporters are less conserved with membrane components in groups of 52 or less and periplasmic binding components in groups of 9 or less.

Mobilization of CaM from nucleus to distal dendrites could be caused by the dissociation of CaM from its nuclear binding components, changes in the permeability of the nuclear pore complex for free diffusion, and/or transported by a carrier.

We identified genes predicted to encode the ABC component (MetN), the permease component (MetP), and the substrate-binding component (MetQ1) in the cluster and found that these genes were downregulated in the mtaR mutant.

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