Sentence examples for binding assay revealed that from inspiring English sources

Competitive drug binding assay revealed that the GNP Chl bind at warfarin binding site I in subdomain IIA of BSA and was further supported by Trp212 fluorescence quenching measurements.

PAMPs binding assay revealed that rCfLec-1 could bind LPS from Gram-negative bacteia and PGN from Gram-positive bacteria, indicating that CfLec-1 could recognize PAMPs and served as PRR.

Furthermore, a solid phase binding assay revealed that HY023016 inhibited ristocetin-induced washed platelets bind to von Willebrand factor (vWF).

Hoechst staining and annexin V binding assay revealed that the compound 4p inhibited tumor cell proliferation through induction of apoptosis.

An in vitro binding assay revealed that 800CW-SCE, 680LT-SCE, and 750-SCE exhibited higher binding affinity than 2-PMPA, which is known as a PSMA inhibitor.

Importantly, immunofluorescence staining and colchcine competitive binding assay revealed that microtubule dynamics was disrupted by 7e by binding at the colchicine site of tubulin.

In vitro binding assays revealed that Syo1 can bind Rpl5 and Rpl11 simultaneously, but also individually.

Hoechst, DCFH-DA staining, mitochondrial membrane and annexin binding assays revealed that the cancer cell proliferation was inhibited through induction of apoptosis in A549 cells.

Resonant mirror detection binding assays revealed that ZFR30 bound to human Band3 with low K(D) (∼ 10nM), and to GPC with higher K(D) (∼ 1nM).

Tissue sections binding assays revealed that Mu-IFN-CSP was also able to specific binding to liver.

In vitro binding assays revealed that cross-linking of Homer dimers enhanced the ability of Homer 1b to bind Drebrin, a known interacting partner.