Endothelial cells express cell adhesion molecules that facilitate rolling, binding, and transendothelial migration of leukocytes [ 13].
Leukocytes can roll on activated platelets enabling binding and migration into inflammatory tissues.
VEGF produced by tumours promotes the proliferation, survival and migration of endothelial cells by binding to its receptors, namely VEGF receptor (VEGFR -1 and VEGFR -1 which and expressed on the endothelial cell surface.
The binding of AT to glucosaminoglycans diminishes the adherence and migration of leukocytes, reduces platelet aggregation and decreases proinflammatory cytokine production [ 15, 16].
Using density-functional tight-binding and ab initio methods we study the adsorption and migration of carbon adatoms on surfaces of single-walled zigzag carbon nanotubes.
Cell binding to FNCC-silk was shown to involve the α5β1 integrin, and to support proliferation and migration of keratinocytes.
The binding energy and migration energy of a vacancy carbon complex are also obtained: the complex is immobile and has higher energy for dissociation than the carbon atom migration energy.
First-principles methods are used to compute the controlling materials properties (vacancy-solute binding energies and migration barrier) for a range of solutes in Al and Mg, and Pauli Electronegativity of the solute relative to the host metal is found useful for correlating properties.
ICAM-2 also facilitates neutrophil binding to and migration through vascular endothelium as a component of immune reactions [ 11, 12].
