Sentence examples for binding activity induced by from inspiring English sources

Acetaminophen inhibited iNOS mRNA expression, promoter activity of iNOS gene and nuclear factor-kappa B (NF-κB) binding activity induced by LPS plus IFN-γ, whereas salicylates did not show any effect on them.

Moreover, the inhibition of NF-κB DNA binding activity induced by genistein was also enhanced by combining genistein with radiation.

Our study particularly shows that the NF-κB DNA binding activity induced by HIV-1 Tat was abolished by the PD98059, a specific inhibitor of ERK.

The analysis of RLU showed that NF-κB p65 DNA binding activity induced by HIV-1 Tat protein at 4 h was significantly increased compared with the controls.

DHMEQ inhibits NF -κB binding activity induced by irradiation and causes an increase in the inhibitory effect on colony formation and in the enhancement of cell cycle arrest in prostate cancer cells.

In contrast to PAO-induced inhibition of nuclear Stat3 DNA-binding activity induced by EGF [6], immunoblotting analysis showed treatment with PAO has no effect on the nuclear presence of EGFR, Src and Stat3 heterocomplex in pancreatic cancer cells (Fig. 3D), suggesting the nuclear presence of the heteromeric complex is not dependent on endocytosis.

Coincubation with indomethacin, a cyclooxygenase inhibitor, had no effect on NF-kappa B DNA-binding activity induced by crocidolite.

In agreement with our hypothesis, CQ co-treatment reverted the p53/DNA-binding activity induced by Zn II), specifically reducing the p53 recruitment on Puma promoter while strongly increasing the p53 enrichment on MDR1 and cyclin B2 promoters.

The diversity in response types detected from this analysis supports a model of PPARβ/δ interacting with chromatin through dynamic binding and interactions; differences in receptor activity induced by endogenous versus exogenous ligands may explain part of this diversity.

As is shown in Fig. 6D, the mutation of binding sites in 3′UTRs of NR2F2 reversed the downregulation on luciferase activity induced by miR-27b, and abrogated the suppressing effect of miR-27b overexpression.

As we reported previously (Lee et al., 2010), binding of the PDZ domain by TMEM88-C antagonized Wnt/β-catenin activity induced by Dvl overexpression.