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The above results suggested that HBV core protein can bind to host gene promoters, especially those involved in tumor pathogenesis.
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Overall, we show here that δ-toxin is able to bind to host AMPs.
IAV hemagglutinin spikes bind to host sialic acids, and viruses enter host cells through receptor-mediated endocytosis.
To date, adhesins are only known to bind to host receptors non-covalently.
Among exogenous MLVs, host range is affected by differences in the viral envelope protein that allow retroviruses to bind to host cellular receptors and enter host cells.
During IAV infection, the viral hemagglutinin (HA) proteins bind to host cell receptors.
LL-37 binds to host DNA [16].
Overall, these data illustrate that δ-toxin directly interacts with, and binds to, host AMPs.
Borreliae are obligatorily bound to host organisms for survival.
The P2 domain is a hypervariable region that binds to host cell [ 13].
The var genes encode P. falciparum erythrocyte membrane protein 1 (PfEMP1)—the major surface antigen of the parasite's blood stages that binds to host endothelial cell receptors to allow the parasite to sequester within various host tissues (Miller et al., 2002; Scherf et al., 2008).
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