Sentence examples for bind to complementary sites from inspiring English sources
MicroRNAs (miRs), as small single-stranded noncoding RNAs, are key post-transcriptional negative regulators that completely or partially bind to complementary sites in the 3′-untranslated-region (3′UTR) of target mRNAs.
miRNAs bind to complementary sites within the 3'UTRs of cognate mRNAs, leading to translational repression or cleavage[4] of the targeted messages.
In animals, miRNAs bind to complementary sites in target mRNAs, generally at 3' untranslated regions (UTRs), to create imperfectly paired miRNA/mRNA heteroduplexes that inhibit translation or increase degradation of target mRNAs.
For animals, the seed sequences of miRNA can bind to complementary sites on 3'UTR of its targeted gene.
For amplification of a band in RAPD analysis same primer has to bind to complementary sites on both strands.
MiRNAs bind to complementary sites on the 3'-untranslated regions (3'-UTR) of target mRNAs to induce cleavage and repression of translation [ 6- 8].
Most plant miRNAs bind perfectly or nearly perfectly to complementary sites on their target genes.
MicroRNAs (miRs) are small noncoding RNAs that bind to complementary/partially complementary sites in the 3' untranslated regions of target genes to regulate protein production of the target transcript and to induce mRNA degradation or mRNA cleavage.
They bind to complementary target sites in mRNA that are often located in 3′ untranslated region (UTR) but can also be found in 5′UTR or even coding region (Forman and Coller, 2010; Li et al., 2011).
They are approximately 18~25 nucleotides in length and can bind to complementary target sites in mRNA molecules, causing translation repression or the cleavage of the targets [ 10]. miRNAs have been reported to play important roles in a variety of pathophysiological processes [ 11– 13].
In mammals, miRNAs are thought to bind to partially complementary sites predominantly located in the 3' untranslated regions (UTRs) of target mRNAs [ 2, 4, 5], thereby enabling the coordinate regulation of genes containing such sites.
