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1: (TD-motifs) The molecules of the miRNA repressor of gene G can strongly bind only at one unique specific site of G- molecules, and once a single such strong bind occurs, the corresponding G molecule degrades extremely fast.
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It is observed that the large bound occurs when (s=n,γ n =1).
The above bound occurs in the case of several delays feeding forward in sequence.
In lines 1 The5 of ASSIGN_HRC, floworstassigned sucase free affinity-mate condition makes equal to zero.
Binding occurs as a result of noncovalent interaction between the receptor and its ligand, at locations called the binding site on the receptor.
Binding occurs in a dose-dependent manner, is saturable, and has an apparent (app) KD 26.53±5.31 nM for monomeric VTNC and 22.62±4.73 nM for multimeric VTNC.
Binding occurs via the UBAN motif within the CoZi domain.
Binding occurs via a domain on DARC known to bind a number of other chemokines.
Binding occurs with either perfect complementarity or, more often, imperfect complementarity [5].
Binding occurs between anionic, sulphated GAG residues and basic amino-acid domains within the chemokine primary structure (Ali et al, 2000).
Binding occurs to the β/β' and σ subunits of RNA polymerase and the nucleotides involved in binding have been determined by footprinting and cross-linking studies [ 3, 5].
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