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This strand-specific mutational bias is explained by the asymmetrical replication.
The link between the uneven distribution of data and the resulting bias is explained by the Yule-Simpson effect.
This results in that a large proportion of variance in codon usage bias is explained by G + C content.
This bias is explained by the increased chance of transfer of a functional unit advantageous to the recipient.
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This bias was explained by either genomic location or an unspecified selection advantage.
Another way in which editors contribute to publication bias was explained by the fact that "we have so many articles coming through... there is an element of pre-refereed selection of papers that never get to a referee".
The observed spin-related movement biases are explained by the model much in the same way as biases during manual catching movements have been explained [16], [17], [24].
In general, synonymous codon usage biases are explained by two alternative but non exclusive models: a neutral mutational-bias and a selective model [ 29].
Increase in the photo-emf response in the fundamental absorption range under bias lighting is explained by this process.
The bias of AncSR1 towards vertebrate ERs is explained by a lack of non-vertebrate sequences used for the reconstruction.
Since protein length and expression levels displayed a strong correlation with codon bias, we tested whether the correlation between protein length and codon bias can be explained by gene expression levels.
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