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In the present investigation, we analyzed urine collected 6 years after the diagnosis to assess the association between urinary markers of nucleic acid oxidation and mortality in patients with established and treated diabetes.
Cox proportional hazards regression was used to examine the relationship between urinary markers of DNA oxidation 8-oxo-7,8-dihydro-2′-deoxyguanosine 8-oxo-7,8-dihydro-2′-deoxyguanosine 8-oxo-7,8-dihydro-2′-deoxyguanosine 8-oxo-7,8-dihydro-2′-deoxyguanosine 8-oxo-7,8-dihydro-2′-deoxyguanosine 8-oxo-7,8-dihydro-2′-deoxyguanosine
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To investigate the association between the urinary markers of DNA (8-oxodG) and RNA (8-oxoGuo) oxidation and mortality in established and treated type 2 diabetes, we used data from the Diabetes Care in General Practice cohort obtained 6 years after diagnosis.
To investigate the relationship between early-onset retinopathy and urinary markers of renal dysfunction.
The dose response assessment in the risk assessment process in humans relies on the relationship between Cd excreted in urine (urinary Cd, U-Cd) and urinary markers of early renal tubular effects (EFSA 2009a; WHO 2011).
We compared urinary [TIMP-2]·[IGFBP7] with two other proposed urinary markers of AKI, urinary NGAL and urinary cystatin C. We aimed to investigate the diagnostic value of these markers to detect AKI within 12 to 48 hours of biomarker analysis.
Urinary markers of nucleic acid oxidation were measured at the Laboratory of Clinical Pharmacology (Rigshospitalet, Copenhagen).
Here we will discuss established, developing and potential serological and urinary markers of renal fibrosis.
The main limitation of urinary markers has been a lack of clarity regarding clinical benefit.
Appointment keeping, presence of urinary markers, and certification by direct observation of treatment.
Table 4 presents results of final linear regression models of urinary markers.
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