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Regional correlation of low- and high-order GSSs between these two markers is indeed intriguing (Fig. 2).
An additional SNP (Lsat_1_v8_lg_4_284,212,481) with 75% match to the rate of deterioration was located between these two markers.
It is therefore likely that the correlation between these two markers could be improved when comparing post-exercise pCort values with sCort sampled with a delay of 10 20 min.
No linkage disequilibrium was apparent between these two markers (Table S4).
The similar distribution between these two markers reveals that most of the amoeboid cells have acquired phagocytic properties.
The interaction between these two markers (SNP rs5770917 and HLA-DRB1*1501-DQB1*0602 haplotype) was then calculated in EHS.
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The frequency of recombination was analyzed between leuA and these two markers to test for independent assortment in the progeny set.
Importantly, although AA and IL-6 both are potent proinflammatory mediators, with AA acting as a substrate for the downstream synthesis of prostaglandins and leukotrienes (via cyclooxygenase and 5-lypoxygenase pathways, respectively), we did not observe quantitative associations between responses based on these two markers.
It was suggested that the FOXL2- and LGR5-expressing pregranulosa cells are two distinct populations because coexpression of these two markers between E13.5 and E18.5 was not observed (128).
However, there was no statistically significant difference in the levels of these two markers between the control and cholangiocarcinoma patients (Mann-Whitney U test; p = 0.057 for CEA and p = 0.056 for CA19-9).
In multivariable analyses, we adjusted for differences in baseline UAE and eGFR between case and control subjects, as these two markers are important risk factors for transition in albuminuria stage.
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