Sentence examples for between the control cell from inspiring English sources

(C) Scanning electron microscopy (SEM) showed the morphological differences between the control cell lines (N1, N2, and N3) and the NS1 over-expressing cell lines (P1, P2, and P3).

This scaling enabled comparisons between the control cell data and the ShB-IR cell data before/after copper treatment.

38 There was a significant difference between the control cell and the 1 ppm dose in the Comet Assay (P < 0.05).

Therefore, in the future, we will use the cDNA microarray and other tools to examine the difference between the control cell and the K -ras transfected cell to understand the cross talks between the Ras-to-MAPK pathway and the regulation of the steroidogenesis process.

The Mann–Whitney U-test, t-test or one-way analysis of variance were used for comparisons between the control cells, the mock-treated cells and the hsa-miR-520d-5p-transfected hsa-miR-520d-5p-transfected hsa-miR-520d-5p-transfected hsa-miR-520d-5p-transfected hsa-miR-520d-5p-transfected

To understand the precise molecular mechanisms of how PI4KIIα regulates EGFR levels, we first analyzed EGFR transcription levels by RT-PCR, and no difference was found between the control cells and PI4KIIα RNAi cells (Fig. S2A and S2B).

Moreover, the 3- 4,5-dimethyl-2-yl -2,5-diphenyltetrazolium bromide (MTT) test showed that cell viability was a little lower in cell groups of N/P 1– 20, but there was no significant difference in cell viability between the control cells and each group of transfected cells with N/P 1 – 20.

Control data (both before/after copper treatment) were scaled by the ratio between the control cells and ShB-IR cells at +30 mV from the previous experiment (larger sample sizes, Fig. 3).

On the homogenous FBN substrate, the migration tracks were compared between the control cells exhibiting calcium oscillations (Fig. 4D) and the cells whose calcium oscillations were inhibited by loading the cells with calcium chelators (Fig. 4E).

To examine the potential role of the IFI16 protein in the glucose restriction-induced activation of the ATM/AMPK/p53 pathway, we knockdown the expression of IFI16 protein in the young WI-38 HDFs (Fig. 5A and B) and compared the activation of the pathway by glucose restriction between the control cells and after the knockdown of IFI16 expression.

After being incubated with 100 μg/ml Lonicera japonica for 4 h and 0.8 J/cm irradiation, many protein spots were found varying in intensity between the control cell-loaded and the photoactivated Lonicera japonica-treated cell-loaded gels.