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No differences between subgroups were identified.
Genes differentially expressed between subgroups were identified using a t-test with a Benjamini and Hochberg multiple test correction (Benjamini and Hochberg, 1995).
Differentially methylated loci between subgroups were identified using Mann–Whitney U-tests, with a P-value<0.05 after Benjamini Hochberg false discovery rate correction (Benjamini and Hochberg, 1995) for multiple testing, with an additional filter that the average change in β between subgroups be >0.2.
Similar(57)
Significant gene expression changes between defined subgroups were identified using t-test in R statistical environment [ 75].
Patients (n=20) from which there were available tumor and matched normal mucosa were grouped into stage (early vs. late) and nodal disease (node positive vs. node negative) subgroups and genes differentially expressed in tumor vs. normal and between the subgroups were identified.
For example, in an approach known as two-phase case-control, the whole case-control dataset was divided into subgroups based on different categories, such as age and gender, before correlations between SNPs and traits in the subgroups were identified.
B; Genes whose expression in tumor was different in normal mucosa and which were most different between the clinical stage and nodal disease subgroups were identified by statistical regression analysis (t test).
Three subgroups were identified.
Four subgroups were identified thanks to our trajectory modeling.
Some common subgroups were identified, such as the proliferation cluster and the beta-catenin cluster.
No sensitive subgroups were identified.
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