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Indeed, enduring neuropathic or inflammatory pain is a well-characterized pathophysiological condition in which a direct parallel between persistent exposure to excitatory/inflammatory neurotransmitters and the increased excitability of spinal post-synaptic neurons has been clearly shown [ 8– 11].
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The stress-age model proposes a linkage between altered birth outcomes through combined effect of chronic medical conditions and persistent exposure to stress, including racism-associated stress [ 5].
But dozens of studies showed that persistent exposure to low levels of ozone damages the respiratory and immune systems.
Conversely, some diseases take time to emerge, e.g. because of persistent exposure to stressful circumstances caused by ambient economic conditions.
In brain scan studies, they have found that with persistent exposure to discordant and fearful stimuli distinct changes occur in the limbic system, particularly the amygdala.
Assuming fairly persistent exposure to respiratory pathogens, a new outbreak would follow relatively soon thereafter.
Little difference in SMs was observed between anti-protein Abs produced by chronic processes (ChI and SAD); both had long CDR-H3s and high levels of SM, and both involve persistent exposure to Ag.
The strong link between earthquake-related exposures and the evolution of PTSD indicates that earthquake exposures may increase symptomatology, the long-term course of posttraumatic stress disorder symptoms may depend on the persistent exposure to stressors [ 30].
Persistent exposure to high concentrations of the proinflammatory cytokines threatened the viability of dopaminergic neurons.
Persistent exposure to ROS initiates the pro-apoptotic pathway of ER stress and caspase activation.
Low-level persistent exposure to asbestos in other populations also has been associated with measurable changes in the lungs.
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