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Besides the prevalence, the age-specific infection patterns were also different between our two studied cohorts.
Interestingly, this latest cluster corresponds to the genes that present the highest expression variation between our two studied conditions (with confirmed qRT-PCRs folds >16) (Table 1).
What caused the phytoplankton difference between our two studied locations remains to be determined.
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The inconsistencies between our two studies regarding behavior autoantibody associations may stem from the increased detection sensitivity afforded by the use of pure antigens rather than the crude fetal brain protein preparations employed in previous studies.
The differences in timing between our two studies may provide additional insights.
This may have caused the difference between our two studies.
However, the calculation of ASE differed between our two studies.
However, there were some important differences between our two studies.
We were therefore surprised to find differences in malaria transmission intensity between our two study sites.
Significant differences in approaches used between our two studies may underlie the distinct conclusions.
We attribute the slight difference in overall glycemia between our two study periods to the surgery-related stress response.
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