Interestingly, Koldewyn and colleagues found a correlation between biological motion perception and ADOS scores, which we did not observe in our sample.
Significant correlations between impaired motion perception and M-pathway dysfunction also point to motion processing deficits in schizophrenia resulting from impaired gain control (28).
Given this, the second major objective of the present research was to explore the relationship between deficits in biological motion perception and social/behavioral outcomes in preterm children.
We were also curious to know whether links between deficits in biological motion perception and "autistic-like" symptoms in preterm children would be stronger for some operations than for others.
The traditional motion coherence paradigm cannot distinguish between local and global limits to motion perception and has hence obscured our understanding of what limits global motion processing during development (and in a variety of neurodevelopmental disorders; Dakin and Frith, 2005).
While identity processing was not the focus of these latter studies, these observations do suggest that further research exploring possible relationships between performance on tests of biological motion perception and both emotional and behavioral outcomes in preterm children is warranted.
One example of such a link between the temporal integration window in motion perception and a neural signal is provided by an electroencephalography (EEG) study, reporting that only 1 EEG spectral component, around 13 Hz, was affected by the continuous wagon wheel illusion (VanRullen 2006).
Additional, exploratory analyses assessed the relationship between scores on each test of biological motion perception and scores on the fives subscales of the AQ (Social Skills, Attention Switching, Attention to Detail, Communication, and Imagination).
We hypothesized that the contrast between static and dynamic actions and their neural processing might shed some light on the neural processing of implicit motion perception and action representation.
This dramatic finding indicates that motion-sensitive areas of cortex by themselves do not suffice for normal motion perception and that additional cortical regions are required to support motion perception.
