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Recent data has indicated a close tie between heart dysfunction and AD.
The association between heart dysfunction and future falls is partly a confirmatory finding, where the most common cardiovascular disorders associated with falls are carotid sinus hypersensitivity, vasovagal syndrome and brady- and tachyarrhythmias [ 46].
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For these reasons, we need more evidence; knowing if this association between primary chronic heart dysfunction and subsequent development of CKD is real.
Both groups with prominent risk for falls had an increased prevalence of heart dysfunction (arrhythmia and ischemic heart disease, respectively).
As troponin mutations are a cause of familial heart disorders in humans, the presence of subtle functional defects as well as quantitative diastolic dysfunction in hdp2 flies suggests homology in heart dysfunction between D. melanogaster and humans.
The results showed acceptable correlation between metrics derived from the two techniques and showed that TPT allowed identification of heart dysfunction in both heart failure and sepsis.
Hyperinflation may play an important role regarding heart size and heart dysfunction in patients with COPD.
For example, cardiomyocyte-specific STAT3 deficiency caused cardiac fibrosis and heart dysfunction with advanced age [ 10].
In addition, echocardiography may further help to discriminate between a systolic and diastolic component of heart dysfunction, which may lead to different therapies.
Myocardial apoptosis and mitochondria damages both contribute to myocardial dysfunction in anthracycline and doxorubicin induced heart dysfunction [ 36– 36].
Valvular heart dysfunction is a significant cause of morbidity and mortality around the world.
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