A link between delayed graft function and chronic allograft injury is evident.
In this study, we examined the association between post-transplant graft function and perioperative xanthine metabolizing enzymes (XME) activity in kidney transplant recipients divided into early (EGF), slow (SGF), and delayed graft function (DGF) groups.
Opposite to our finding, Gupta et al. [ 10] have reported that acute rejection episodes, delayed graft function, and one-year graft survival was similar between patients with and without HLA specificities defined by solid phase assay.
Episodes of delayed graft function and the number of biopsies for cause were similar between the New and the Previous KAS.
Pratschke, J., Weiss, S., Neuhaus, P. & Pascher, A. Review of nonimmunological causes for deteriorated graft function and graft loss after transplantation.
It causes delayed graft function and graft loss in long-term follow-up studies.
The ensuing impairment of graft survival makes urgent the need for further elucidation of delayed graft function and a search for an effective therapy.
We also compared the control kidneys and KNW with regard to prompt function or delayed graft function and satisfactory versus unsatisfactory function (unsatisfactory: serum creatinine > or =2.5 ml/dl or graft loss at 6 months) to identify donor and recipient characteristics associated with delayed graft function and unsatisfactory outcome.
Other variables hypothesized as having a relationship to initial graft function and patient and graft survival were also extracted.
