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When CEA was high, differences between genotype groups failed to reach significance.
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There was no difference between genotype groups.
We also compared weight between genotype groups.
Treating APOE genotype in this fashion yields a monotonic relationship between genotype group and risk of CHD [4].
Again, the difference between groups failed to be significant (p = 0.5048).
Although the MRs were decreased by quercetin treatment in the two genotype groups, we failed to find any significant difference in the inhibition degree on MR between the two genotype groups, neither on the Cmax and AUC(0 24 h) reduction degrees of paraxanthine, indicating that CYP1A2 genotype had similar or no effect on quercetin treatment.
The difference between the two age groups failed to reach statistical significance, F 1,34) = 1.4, p>.2.
Therefore our hypotheses of difference in treatment effect between treatment group and placebo group failed.
Interestingly, however, there were no significant differences between the genotype groups with regard to this modulation.
Independent t-tests were used to analyze differences between the genotype groups for STAI-data.
As expected, cholesterol levels differed significantly between the genotype groups.
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CEO of Professional Science Editing for Scientists @ prosciediting.com