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Table 1 summarises the relationship between expression of total cellular activated c-Src and clinico-pathological factors.
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No significant difference was found between the expression of total HIF-1 α and hydroxylated HIF-1 α.
We have also noticed that no significant difference was found between the expression of total and hydroxylated HIF-1 α induced by HBx.
We found that there is no difference in the expression of total GluR1 between wild-type (n = 4 mice) and neurabin KO mice (n = 4 mice, P>0.05, paired t-test; Fig. 4E and G).
However, we found no significant differences in expression between individuals of different genotypes for expression of total TSFM and TSFM WT.
There is no alteration in expression of total VE-cadherin between control and infected experimental monolayers at different time p.i. as detected by Western blot assay.
Pearson's correlation analyses demonstrated significant inverse correlations between the expression of miR-574-5p miR-574-5p miR-574-5p totandQki mRNA (Pearson r=−0.4082, p<0.05) or QKI prothen (Pexpression0.4406, p<0.05) in the 60 pairs of CRC samples totald (figure 7D,E).
To explore the relationship between the expression of miR-574-5p miR-574-5p miR-574-5p totandQki mRNA/prothen, wexpressiond qPCR and western blofs witotal pairs of clinical samples.
This finding reflects the most striking difference between the two patient cohorts, which was CD19 expression of total plasma cells.
The cardiac left ventricular expression of total CaMKII was not different between genotypes and/or treatment groups (Fig. 6 top).
Except for the sensitive H2347 cell line, which showed lower expression, the expression of total FOXO3a was not noticeably different between the sensitive and resistant cell lines.
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