The age- and sex-adjusted correlations between complement components are presented in Table 3. C3, factor B, properdin, and factor H were all significantly correlated with each other in SARs and SACs.
We could not find the correlation between BMI, disease activity, 25(OH D, ESR, CRP, complement components, and low BMD at the lumbar spine and at the hip.
Linear regression analysis was carried out to identify independent predictors of individual complement components, and general linear model analysis was carried out to determine whether differences in complement components between relatives and control subjects were independent of covariates.
Other complement components and immunoglobulins were absent.
Various studies in animal models and clinical studies have demonstrated elevated levels of complement components and complement activation fragments in serum, cerebrospinal fluid (CSF), and brain parenchyma after TBI, with broad parenchymal deposition of complement components [ 6- 9].
Measurements of serum complement components and serum immunoelectrophoresis were within normal limits.
Immunologic measures were regarding complement components and anti-double stranded DNA antibodies (anti-dsDNA antibodies).
Furthermore, multivariate analysis revealed the relationship between the U-complement components and tubular damage markers.
During disease flare in SLE, a strong positive correlation occurred between COMP-C3b and complement components C1q and C4, but no correlation between COMP-C3b and C3.
