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Moreover, both methods were identical for the positive and negative directions of all the genes between both time points (File S1).
Prescriptions for psychotropic medications changed very little between both time points.
Models regarding comparisons between both time points excluded those subjects at baseline who did not participate at t2.
A reduction of −40.9% (p < 0.05) in ICG enhancement between both time points was observed using iloprost.
Retest-reliability was computed by means of the Pearson-correlation coefficient of the subscale scores between both time points.
Firstly, patients expressed pre-occupations and feelings about ICTS that changed between both time points, such as fear of side effects at TP1 that diminished at TP2.
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Hence, the differences between all temperature treatments for a given time point were much smaller than between temperatures for both time points.
A lack of overlap between genes at both time points was observed, with only AKIP being differentially expressed at both time points.
The cortisol:DHEAS ratio differed between the groups at both time points and mediated the relationship between depression and slow walking speed at month 6.
The correlation between HWB scores at both time points was excellent (r = 0.90), with no significant differences between mean scores or variance of the data sets.
There were no differences in bacterial loads in lung, blood or spleen between the groups at both time points.
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Justyna Jupowicz-Kozak
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