In contrast, nominally better visual memory (0.13) and learning (0.12) is present.
Higher delayed memory on Logical II was associated with lower depressive symptoms (GDS; β −0.22, P<0.05) and better visual memory with lower stress (DASS-stress; β −0.35, P<0.001), depressive symptoms (DASS-depression; β −0.28, P<0.05), and anxiety symptoms (DASS-anxiety; β −0.24, P<0.05).
Correlational analysis showed that patients with right hippocampal sclerosis with better visual memory activated the amygdala bilaterally, right anterior parahippocampal gyrus and left insula.
In right temporal lobe epilepsy, greater right hippocampal activation for face encoding correlated with better visual memory.
In patients with right hippocampal sclerosis, insula activation was associated with better visual memory implying efficient recruitment.
Patients with right hippocampal sclerosis with a better visual memory showed significant correlations with activation within the left amygdala in addition to activations within the right anterior parahippocampal gyrus and left insula that were seen in the visual subsequent memory analysis (Table 9).
In the posterior medial temporal lobe there was a significant positive correlation between memory asymmetry indices for encoding faces and change in design learning scores after right ATLR, characterized by greater right than left posterior medial temporal lobe activation being correlated with better visual memory outcome (R2 = 0.16, P = 0.05) (Fig. 4).
In right temporal lobe epilepsy, greater right than left anterior hippocampal functional magnetic resonance imaging activation on face encoding predicted greater visual memory decline after right anterior temporal lobe resection, while greater right than left posterior hippocampal activation correlated with better visual memory outcome.
PAL assesses visual memory and new learning.
27 28 These included weakness in visual object recognition, visuospatial skills, visual memory and oculomotor control.
