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We conclude from these comparisons that the observed network structure is not better explained by random processes.
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In State 1, receptor activity, integral to membrane, and signal transduction are the only GO annotations in which the gene expression levels are better explained by histone methylation levels (than random selection of genes).
The tandem-duplication-random-loss model, however, cannot account for inversions and transpositions, which may be better explained by nonhomologous, intrachromosomal recombination.
These variables are probably better explained by others minor components.
This delayed improvement in oxygenation is better explained by slowing of disease progression.
The performance improvement can be better explained by examining Figure 5b.
The adsorption both physisorption and chemisorption is better explained by the Freundlich model (Ho and Wang 2008).
The hysteresis can be better explained by the enhanced stability of active sites under reaction conditions.
Time-dependent variation in resighting rates was better explained by local rainfall (45.1% variance explained) than by the NAO (33.5% variance explained, Figure 1a).
Therefore the relatively low recapture rate is better explained by dispersal and mortality.
Furthermore, we demonstrate that our results are not better explained by an oscillatory entrainment mechanism.
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