Sentence examples for beta has been shown from inspiring English sources

The efficacy of interferon (IFN) beta has been shown in several placebo-controlled, parallel-group studies in relapsing remitting multiple sclerosis (RRMS).

BetA has been shown to induce apoptosis independently of the extrinsic pathway [28].

A beta has been shown to selectively inhibit the chymotrypsin-like activity of the 20S proteasome leading to formation of inclusion bodies in neurodegenerative disorders such as Alzheimer's disease and Down's syndrome [5], [6].

Estrogen receptor beta has been shown to be a physiological inhibitor of bone growth [ 39].

Although there are currently limited long-term, controlled effectiveness data, ERT with agalsidase alfa or beta has been shown to reduce the decline in renal function in short- and long-term studies in male, female, and paediatric patients.

ER beta has been shown to inhibit human breast cancer cell proliferation by repressing transcription of the c-myc, cyclin D1 and cyclin A genes and increasing the expression of the cyclin-dependent kinase inhibitors p21Waf1/Cip1 and p27Kip1, leading to cell cycle arrest in the G2 phase [ 5, 40, 61].

Since 1993, the beneficial effects of interferon beta have been shown [ 1], and in Japan, interferon beta-1b (IFN beta-1b) has significantly reduced relapse rates and reduced MRI lesion areas in patients with relapsing-remitting MS [ 2].

Moreover, S100-beta has been shown to fail to identify patients with lymph node micrometastases detected by sentinel node procedure [ 1].

Serum S100-beta has been shown to be superior compared to MIA, as an early indicator of tumour progression, relapse or metastasis [ 18, 43], and its distribution as a serum biomarker of melanoma, therefore, is the broadest [ 36].

In melanoma cells, TGF-beta has been shown to significantly inhibit melanin synthesis by increasing the rate of degradation of tyrosinase, a rate-limiting enzyme in the melanogenesis pathway [ 35, 36].

Secretion of various cytokines such as IL-1-beta, IL-6, IL-18, monocyte chemotactic protein-1 (MCP-1), regulated upon activation normal T cell expressed and secreted (RANTES), macrophage inflammatory protein-2 (MIP-2), intercellular cell adhesion molecule-1 (ICAM-1), and transforming growth factor beta (TGF-beta) has been shown to be increased in cisplatin-induced AKI [ 79].