By doing so, we aimed to better answer whether cuing event structure per se benefited memory encoding.
This could give an opportunity to process event structure as such, but just as easily could benefit memory by allowing one to process other aspects of the activity such as objects or goals.
Remindings specifically benefit memory for the first instances in related pairs, which suggests that the large mnemonic consequences of remindings arise from retrieval of the first presentation during encoding of the later related episode.
As opposed to disrupting memory for the original event, as proposed by Chan and LaPaglia (2013), the results of the present study indicate that interim testing may benefit memory for the original event, and tentatively suggest that interim testing may improve discrimination between original and post-event details.
Since overexpression of NR2B is reportedly capable of enhancing memory function in mice [7], [20], [21], we wonder whether overexpression of NR2B would also benefit memory function in rats.
A single dose of methylphenidate has been shown to benefit memory in healthy subjects, as well as increase subjective ratings for interest, excitement and motivation in a mathematical task.
Thus, based upon the experimental literature, in a population of healthy fit older individuals, increased levels of ghrelin may benefit memory performance and may be related to body composition.
Although the isomaltulose-based meal benefitted memory (Fig. 2) and mood (Fig. 4), regardless of the day on which it was consumed, there were also effects observed only on the second day of testing.
Contrasting these direct effects of insulin on central nervous system (CNS) function, reduced peripheral insulin sensitivity has been linked to subtle cognitive deficits and reduced spontaneous cortical activity in otherwise cognitively healthy humans (7, 8), suggesting that the ability of insulin to benefit memory functions is attenuated when peripheral insulin signaling is disrupted.
The direction of change in the expression of the five genes should tend to cause excitatory synaptic plasticity in neural cells and/or benefit memory ability (although gene MECP2 locates in X chromosome, the equal sex ratio for both the domesticated and wild groups should eliminate the sex-linked effects on dosage).
