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Although TRF1 is believed to regulate telomerase in cis at individual telomeres, TRF2 may control telomere length by telomere degradation (Ancelin et al., 2002).
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In rodents, clock genes drive circadian rhythms in white blood cell function, and glucocorticoids are believed to regulate these rhythms.
Each miR is believed to regulate multiple genes.
p38 is believed to regulate both apoptosis and autophagy.
Cxs have long been believed to regulate chemoresistance by exerting GJIC.
Now, Sirt1 is believed to regulate apoptotic thresholds by deacetylating p53 [ 6, 12].
Discrete and independent circadian and homeostatic processes are believed to regulate sleep.
Cxs have long been believed to regulate tumor development during carcinogenesis by exerting GJIC.
Myc is believed to regulate expression of 15% of all genes [ 77].
There, it is believed to regulate the ER machinery responsible for Ca2+ release.
P73α is known to bind to RACK1, whereas p73β and p53 do not, and both p73α and p73β were reported to regulate telomerase activity [23].
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