Cells were treated with each transition metal at a concentration of 1 mM for 48 h and viability was determined.
Cells were treated with each transition metal (in nominal µM: Cu 80; Co 400; Zn 31.2) alone or in the combination with Ag (15 µM), for 24 h.
When A549 cells were treated with each Tat-conjugated peptide, the peptides penetrated the cells and EGF-stimulated tyrosine phosphorylation of EGF receptor was significantly suppressed.
When breast cancer cells were treated with each of these synthetic Tat-conjugated peptides, the conjugated peptides penetrated into the cells and suppressed cell proliferation.
KN63 cells were treated with each test chemical and gene mutations were detected in the gpt gene of the shuttle vector rescued from the KN63 cell genome into an E. coli host.
Initially, exponentially growing cells were treated with each agent alone to determine effective doses.
Following induction of protein expression with isopropyl-beta-d-thiogalactoside, the cells were treated with each drug for 3 h (1 h for 4-hydroperoxyifosfamide).
Dye solutions of a series of concentrations (0 1000 ppm) was treated with each biosorbent in 1 500 solid/solution ratio, and the optimum shaking and settling time periods were allowed for each system to reach equilibrium.
Five hours after transfection, the cells were treated with each compound concentration for an additional 24 h, and the luciferase assay (Promega) was performed according to the manufacture's protocol.
Twelve pellets were treated with each culture, plus a negative control (water only).
