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The next question was obvious: what is the mechanism behind this modulation of endocytosis by the number of action potentials?
Chimerel et al. demonstrate that indole modulates the hormone secretion of enteroendocrine L cells and reveal the molecular mechanism behind this modulation.
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Recent research suggested that this modulation takes place by a modulation from the TPJ8.
This modulation is analogous to AM radio.
However, the neuronal circuits that underlie this modulation remain unknown.
Instead, this modulation requires further processing stages.
Unfortunately, this modulation resulted in a elevated basal level which under non-induced conditions attained the output of induced pJOE4056.2 cells, though it evidently fell behind in expression compared to pJOE5058.1 (data not shown).
Are they behind this?
At larger modulation frequencies, both devices show increasingly negative phase shifts due to charge carriers lagging behind the modulation frequency.
As the modulation frequency increases past the maximum, IMPS images lose contrast owing to carriers lagging behind the modulation.
Finally, in Experiment 8 we showed that search for interocular conflict is about optimal when the display is present for just 150 ms, supporting our hypothesis that abnormal fusion is the driving force behind the modulation of conspicuity of interocular conflict.
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