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The possible reasons behind this effect have been discussed.
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The mechanism behind this effect has been explored by several studies.
SiN has been demonstrated in cross sectional and longitudinal studies, but the mechanisms behind the effect have yet to be proven.
However, despite these convincing data, the mechanisms behind this protective effect have not yet been established.
The mechanisms behind this cardioprotective effect have been attributed to the many positive effects that β-blockers have on cardiac function: antiarrhythmic effects, improvement of myocardial function, lowering of cardiac oxygen consumption, and lowering of blood pressure.
However, the mechanism behind this protective effect has not been established.
The mechanisms and processes behind these effects have been investigated and described.
Hypotheses behind these effects have been linked to the generation of ceramide and to the inactivation of PAF [ 11, 24].
The specific mechanisms behind these effects have not fully elucidated yet, but many scientific researches suggest that n-3 PUFAs may act at several levels.
The n-3 LCPUFAs are also suggested to have antitumour effects including inhibition of angiogenesis and metastasis (Baracos et al, 2004); however, the specific mechanisms behind these effects have not been elucidated.
In this paper the possible mechanisms behind the solids effect have been divided into the following four categories: Compositional and substrate effects; product inhibition; water concentration; and cellulase adsorption.
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CEO of Professional Science Editing for Scientists @ prosciediting.com