Sentence examples for behavioural defect from inspiring English sources

To investigate the causes of the behavioural defect further, we analysed inner ear morphology in the rescued swr adults by histological sectioning.

Thus, expression of CRH-1 in AFD restores the behavioural defect of crh-1 tz2) crh-1 tz2

One could argue that our locomotor models may not be so sensitive to detect this kind of behavioural defect.

We now provide evidence that restoration of C. elegans CREB orthologue in one neuron recovers the CREB-dependent behavioural defect.

The behavioural defect of crh-1 tz2) crh-1 tz2esemutantshat of AFD-ablated animals, as resemblespreviously by thatqualitative single-wofm assay (Mori & Ohshima, 1995).

Although CRH-1 is broadly expressed (Kimura et al, 2002), expression of CRH-1 only in AFD of crh-1 mutants recovered its behavioural defect, whereas expressions in other neurons that comprise the thermotactic neural circuit and in the ASH polymodal neuron did not rescue the behavioural defect of crh-1 tz2) crh-1 tz2

Braungart et al. [54] performed a focused compound screen using the C. elegans MPTP model of PD and found that lisuride and apomorphine (dopamine receptor agonists), as well as rottlerin (protein kinase C inhibitor) ameliorated the MPTP-induced behavioural defects when present at a low concentration.

In contrast, in the adult rescued swr fish, we see a gross structural abnormality in the inner ear that is robust, highly specific and consistent with the observed behavioural defects.

In addition to the reported behavioural defects of casy-1 mutants, we show here that the pan-neuronal expression of human CLSTN2 rescues the chemotaxis conditioning defect of casy-1 tm718 casy-1 tm718sthusing a strong conservation between CLSTN2 andemonstratinghe level of their molecular function.

We report here that high doses of genistein aglycone, given continuously over a 9 month period to MPSIIIB mice, significantly reduce lysosomal storage, heparan sulphate substrate and neuroinflammation in the cerebral cortex and hippocampus, resulting in correction of the behavioural defects observed.

This limited pathogenicity of DWV and VDV-1 contrasts with the strong association of DWV incidence in worker bees with winter mortalities of colonies [51] [53] and the induction of the typical deformed wing pathologies and drastically reduced life expectancy in workers bees [30] as well as sublethal behavioural defects though infection of the brain tissues [54] [56].