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This approach would thus generate the prerequisites for a deeper understanding of the behaviour of this pathogen in its human host.
These findings help to provide a better understanding of the evolutionary behaviour of this pathogen.
There has been a controversial suggestion that infection by Salmonella may subsequently cause an increase in mortality for up to one year [ 1], and it is clear that we need to improve our understanding of the behaviour of this pathogen.
The next challenge is to define which local nutrients are assimilated by C. albicans cells during disease establishment and progression, and how this influences the behaviour of this pathogen in the host.
Despite this, the behaviour of this pathogen in the marine environment and how it bioaccumulates in shellfish have not been investigated, although the public health risk associated with the consumption of shellfish contaminated with other human enteropathogens is well documented [ 12].
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the behaviour of the pathogen in a host and its cells.
Often, the observations made in outbreaks suggest further specific applied research to determine the behaviour of the pathogen in that specific matrix, handled in a specific way.
The regulatory influence of the CREE and the copy number differences between N. gonorrhoeae, N. meningitidis, and N. lactamica may contribute to the different behaviours of these pathogens through differences in their regulatory networks.
This, however, requires sound knowledge on the behaviour of both pathogens in water.
Many factors are important in shaping the epidemiological and evolutionary behaviour of pathogen populations in space and time.
With the aim of decreasing listeriosis occurrence, the early detection of this pathogen in food products is of tremendous interest.
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Justyna Jupowicz-Kozak
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