This behaviour has been attributed to the fact that the virtual gluons that flit between the quarks within a hadron are not neutral but carry mixtures of colour and anticolour.
The reason for this difference in behaviour has been attributed to the more compact nature of the β-polymorph unit cell, when compared to that of the α-polymorph.
Whereas the normal slow crack growth behaviour has been attributed to the disentanglement of the molecules by sliding without chain scission, it is proposed that the abnormal fracture behaviour may involve a disentanglement mechanism that consists of a combination of chain sliding and scission.
This behaviour has been attributed to two phenomena, occurring concomitantly: that of macromolecular chain orientation causing a reduction of free volume and a (relative) increase of Tg, and chain scission caused both by chain elongation and thermal degradation, leading to a drop of Tg.
Stromal cells do not contact luminal cells directly in vivo because they are separated from them by basement membrane, and in some cases the effect they have on epithelial cell behaviour has been attributed to the production of ECM proteins [44].
Shape change and highly motile behaviour associated with high Prox1 expression have been attributed to a Prox1-mediated upregulation of integrin α9β1 (Mishima et al, 2007), which can also function as a receptor for VEGF-C and D (Vlahakis et al, 2005).
The differences have been attributed to occupant behaviour and poor quality of the construction.
These larger discrepancies have been attributed to the lack of caregiver awareness of problematic behaviours and inconsistency in symptom manifestation across contexts (e.g., home, school) [ 13].
