Sentence examples for behavioral phenotypes of the from inspiring English sources

Methods available for quickly and objectively quantifying the behavioral phenotypes of the fruit fly, Drosophila melanogaster, lag behind in sophistication the tools developed for manipulating their genotypes.

The electrophysiological and behavioral phenotypes of the GluR1 (S845A/S831A) mutants are quite similar to the phenotypes we observed for the AKAP5 D36 mutants, and is consistent with results showing that inhibitors of all PKA/AKAP interactions can disrupt synaptic plasticity and certain forms of spatial memories [58].

The major ones are: 1) We are told nothing about the behavioral phenotypes of the 12 lines at the elevated temperature.

We found similarities in the cellular/molecular, immunohistochemical, electrophysiological, and behavioral phenotypes of the α-CaMKII hKO and pilocarpine-treated mice.

In order to examine the biological roles of the brain-expressed FABP genes, we characterized behavioral phenotypes of the relevant KO mice.

The similarity of the anatomical and behavioral phenotypes of the Jna/+ mice to the Dcx, Lis1, and reeler mouse mutants led us to consider whether mutations in the human homolog of Tuba1 might cause lissencephaly in humans.

A recently identified deficit in inhibitory serine-phosphorylation of GSK3 in Fmr1 knockout mice raised the possibility that dysregulated GSK3 contributes to some of the behavioral phenotypes of these mice [20], [21].

Although maternal behavior is likely influencing behavioral phenotype of the infant independent of the physiological transfer of milk (Champagne 2014; Dettmer et al. 2014), inferences from breastfeeding and formula-feeding mothers suggest that glucocorticoids ingested via milk are more predictive of infant behavioral phenotype than are maternal circulating glucocorticoids (Glynn et al. 2007).

Hanson E, Bernier R, Porche K, Jackson FI, Goin-Kochel RP, Snyder LG et al. The cognitive and behavioral phenotype of the 16p11.2 deletion in a clinically ascertained population.

The strength of behavioral genetics and the mouse models is the ability to apply behavioral paradigms known to be subserved by known anatomical loci not only to determine the behavioral phenotype of the model, but also to elucidate candidate brain regions affected by the mutation.

Here we present a thorough behavioral phenotyping of the TS2-neo mouse, capitalizing on this unique opportunity to use the TS mutation to model ASD in mice.