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Quercetin alleviated behavioral dysfunction in chronically stressed animals.
Therefore, current study was designed to investigate the effect of quercetin on chronic unpredicted stress (CUS) induced behavioral dysfunction, oxidative stress and neuroinflammation in the mouse hippocampus.
Since FTLD is characterized by various emotional deficits, it would be helpful to establish a marmoset model of FUS-silencing in various brain tissues for investigating the pathomechanism of higher cognitive and behavioral dysfunction.
We tested whether indomethacin would have an effect on cognitive and behavioral dysfunction, as well as dysfunction of the activities of daily living, in patients with mild to moderate AD.
HAD, the most severe form of HIV-1 induced CNS impairment [6], is clinically characterized by motor and behavioral dysfunction that in the absence of therapy may lead to seizures, coma, and death within six months of onset [3].
This deterioration is characterized clinically by cognitive and behavioral dysfunction [ 197].
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Physical impairments and cognitive, emotional, and behavioral dysfunctions present significant challenges when TBI patients are discharged and return to their respective home environments and social communities.
Although, in general, the early detection of risk factors indicating a development of behavioral dysfunctions may lead to negative social reactions, this relation is probably stronger for neurobiological risk factors, since people often believe that neurobiological characteristics are static or deterministic (Horstkötter et al. 2012; Walsh 2009).
These results presented a temporal evaluation of phenotypic behavioral dysfunctions in IDUA−/− mice from adolescence to maturity, indicating the impairments, with different ages of onset, in locomotor and anxiety-like compulsive behaviors, spatial learning and memory, visual recognition and short-term non-associative memory retention.
Moreover, this virus causes mental and behavioral dysfunctions in children that have been infected in utero [1], [2].
Prenatal PolyI C exposure in mice and rats is known to induce a wide spectrum of brain and behavioral dysfunctions implicated in schizophrenia and/or autism (for a review see [36]).
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