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In keeping with this observation, several circadian behavioral defects were observed in the CD38-KO mice.
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Behavioral defects are observed in RSK2 knockout mice and in Drosophila upon knockout of RSK, the single fly ortholog of vertebrate RSK proteins.
Obvious defects were observed in seven of the 18 mutant strains.
Similar defects were observed in Rock2b depleted embryos.
Similar but milder defects were observed in aged hearts.
Defects were observed in the particle structure.
A significant body weight reduction and neurological defects were observed at D3 after SAH.
No germination defects were observed.
However, no apparent anatomical defects were observed in these mice.
No gross somatic defects were observed in adult Tex19.1−/− mice.
Two defects were observed in prophase and early prometaphase cells.
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