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The compressive behavior of the cellular concrete utilizing millimeter-size spherical saturated SAP with different porosities was experimentally investigated under quasi-static and high strain-rate loadings.
Drop tower tests were conducted to characterize the mechanical behavior of the cellular TIMs made of an intrinsically brittle base material.
Good agreement on structural eigenfrequencies between the effective continuum solutions and the exact solutions shows that the proposed continuum model can accurately simulate the dynamic behavior of the cellular solids.
The results of our model are consistent with the behavior of the cellular slime mold Dictyostelium discodeum, which switches from a single-celled to a multi-celled state when resources become limited.
We show that the rigid-perfectly-plastic-locking model cannot predict shock formation at a material interface, so we propose an elastic plastic-densifying modelastic plastic-densifyingstrain behavior of the cellular modelials.
The behavior of the cellular transport of Tat peptide-conjugated PS-PEG micellar QDs is consistent with that of Tat peptide-conjugated QDs previously reported in the literature [42].
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By adding more network influencing accessory proteins, one commonly expects to gain a better understanding of cytoskeletal behavior on the cellular level.
The underlying fundamental mechanisms governing the compressive and fatigue behavior of the graded cellular structures are elucidated via in situ tomography experiments and digital volume correlation analyses.
In addition, the whole structure and dynamic behavior of the multilayer cellular NNs are seriously dependent on the evolution time of each variable and its position (space), but also they are intensively dependent on its interactions deriving from the space-distributed structure of the whole networks [23].
These findings suggest that mechanical strain, or another closely related variable, may influence the design and ultimate mechanical behavior of the extra-cellular matrix found in lamellar bone.
We report here the utilization of a microarray covering the whole genome of Staphylococcus aureus to evaluate the behavior of the bacterium upon cellular internalization, a phenomenon potentially contributing to bacterial protection against host defenses, and important in the context of chronic infections.
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