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The four-week-old male mice were injected intravenously with phosphate-buffered saline (PBS, pH 7.4) or TNF-α (6 µg/kg) via the lateral tail vein 6 hrs before intraperitoneal administration of DEN (10 mg/kg).
before intraperitoneal administration of 5-HTP (200 mg kg−1).
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Additionally the number of intraperitoneal tumor cells was counted before and after intraperitoneal administration of catumaxomab but no imaging method was used to assess the response to catumaxomab [ 6].
Cadmium toxicity (LD50) was determined in different age groups of animals treated with ash for five weeks before a single oral or intraperitoneal administration of cadmium chloride.
For experimental wounding, the back of the animals was shaved 48 h before the procedure under general anesthesia induced by the intraperitoneal administration of ketamine (1.0 ml/kg) and xylazine (0.2 mg/kg).
Before debridement injury, 3-month old mice were anaesthetized by intraperitoneal administration of Avertin at 0.45 mg/g body weight (2, 2, 2-tribromoethanol, Sigma-Aldrich).
Each animal received intraperitoneal administration of fluorochrome calcein (20 mg/kg) 24 h before autopsy.
One day before the start of treatment, CD4+ T cells were depleted by intraperitoneal administration of the CD4+ T cell depleting mAb GK1.5, and GK1.5 treatment was continued weekly until the end of follow-up.
Intraperitoneal administration of kainic acid transiently induced the c-fos mRNAs in medaka brains.
Intraperitoneal administration of pentoxifylline (200 mg/kg) abrogated this effect.
Intraperitoneal administration of fucoxanthin significantly inhibited the growth of tumor mass in B16F10 cells implanted mice.
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