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Blood samples were collected by jugular venipuncture before administration of either stevia or karo and at 60 and 240 min after administration.
The mice were pretreated with glibenclamide (10 mg/kg), an ATP-sensitive K+ channel inhibitor, intraperitonially 15 min before administration of either diclofenac sodium or MECN.
The mice were pretreated with glibenclamide (10 mg/kg), an ATP-sensitive K+ channel inhibitor, intraperitonially 15 min before administration of either diclofenac Sodium or MEPH.
To verify the possible involvement of cGMP pathway in the antinociceptive action caused by MEPH the mice were pre-treated with methylene blue (20 mg/kg), a non specific inhibitor of Nitric oxide/guanylyl cyclase, intraperitonially 15 min before administration of either diclofenac sodium or MEPH.
Treatment of rodents with blockers of AT1 (ARBs) before administration of either MPTP or 6-OHDA prevents neurotoxic effects of these agents through less microglial activation, less generation of NADPH oxidase (e.g., Nox4) and therefore less superoxide production, and less neuronal dysfunction and loss, suggesting that ARBs promote dopamine neuron resilience and survival.
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The effects produced by propofol or remifentanil on discharge activity of subthalamic neurons were studied intraoperatively to determine whether they alter neuronal activity.Microelectrode recordings from 11 neurons, each from individual patients, were discriminated and analyzed before and after administration of either propofol or remifentanil.
15 min before oral administration of either vehicle or MEMC (500 mg/kg).
We monitored thermal and mechanical sensitivity before and after administration of either LPA (i.t).
Ten women with IBS (mean age 30±10 years) and ten control subjects (all women) (mean age 29±7 years) rated pain intensity and unpleasantness to distension of the rectum (35 mmHg) and thermal stimulation (47 °C) of the foot before and after rectal administration of either lidocaine jelly or saline jelly in a double blind crossover design.
Withdrawal latency (reaction time of the animal when placed on a surface heated to 52 °C) was measured before and 30 min after oral administration of either saline tramadol (2 mg.kg−1) or ASE (30, 100 or 300 mg.kg−1).
The validity of Section 3501 had not come before the court until now because no administration, of either party, sought to apply it.
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