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The tibia and patella have been targeted for bone lead research because they consist mainly of cortical and trabecular bone, respectively.
The tibia and patella have been targeted for bone lead research because these two bones consist mainly of pure cortical and trabecular bone, respectively.
The tibia and patella have been targeted for bone lead research because these two bones are primarily cortical and trabecular bone, respectively, with different ramifications in terms of toxicity (Hu et al. 1998).
The tibia and patella have been targeted for bone lead research because they consist mainly of pure cortical and pure trabecular bone, respectively, and thus represent the two main bone compartments.
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Internal devices are normally implants that are targeted for bone, tissue and tissue fluid, and blood.
The tibia and patella were targeted for bone lead research because they consist mainly of pure cortical and pure trabecular bone, respectively, and thus represent the two main bone compartments.
After preliminary studies established the onset of disease between 10 and 12 weeks of age, 6- to 8-week-old male W/BF1 mice were targeted for transplantation with either T-cell-depleted bone marrow or purified hematopoietic stem cells from haploidentical B6C3/F1 mice.
These sites are targets for bone lead research, because the half-life of lead in tibia and patella differs.
These sites are targets for bone lead research because the tibia consists mainly of cortical bone, and the patella of trabecular bone.
We believe Col10a1 trans-activators are promoters of chondrocyte hypertrophy and are targets for low bone growth as seen in skeletal dysplasia, whereas repressors will decrease Col10a1 expression and delay chondrocyte hypertrophy and are, therefore, targets for bone over growth as seen in osteophyte formation in osteoarthritis.
Treatments for osteoporosis could be targeted toward increased bone mineral density, improved architecture, or repair and prevention of microdamage.
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