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It has previously been suggested that modulation of cell stress pathways can alter the response of neurons to neurodegenerative stimuli [13] [14].
It has been suggested that modulation of sympathetic activity on the heart is best estimated when normalized to total power (PLO/PTOT) rather than PHI [ 47].
It has been suggested that modulation of tumour cells, either by specific AQP inhibitors/blockers and agonists or by indirect modulation of closely associated growth factors, may become a future treatment strategy for some tumours [ 24, 25].
On the basis of recent structural analysis of ERGIC-53, also known as LMAN1, it has been suggested that modulation of binding by different Ca2+ concentrations occurs in various luminal compartments in cells [ 24 ].
Moreover, it has been suggested that modulation of keratins due to Epithelial-to-Mesenchymal Transition (EMT) occurs frequently in CTCs of breast cancer patients and may be associated with an unfavorable outcome [ 1].
For instance, it has been suggested that modulation of cellular activities of the forkhead transcription factor FOXQ1 may have an application in cancer therapy since its inhibition blocks epithelial to mesenchymal transition and results in cancer cell sensitisation to a variety of chemotherapeutic agents (Qiao et al, 2011).
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While a chronic foreign body response should be avoided to prevent impedance of tissue regeneration by inflammation and fibrosis (characteristics of chronic foreign body response), it is suggested that modulation rather than avoidance of immune response is more desirable for tissue remodeling, which should be taken into consideration for developing immunoisolation strategies.
Since it was suggested that modulation of Bmi-1 protein might be involved in human colorectal carcinogenesis by repressing p16Ink4a/p14ARF proteins {14}, we further examined the expression of p16Ink4a/p14ARF by IHC in our ovarian carcinoma cohorts.
Accordingly, albeit further investigations are required, achieved results are suggesting that modulation of the TME through the targeting of IL-1 activity combined with currently used chemotherapeutic agents might constitute a novel efficient anti-tumoral strategy for treating mutp53 carrying tumors.
It has been suggested that the modulation of local EGF production is directly involved in the pathogenesis of tubular damage.
Furthermore, it has been suggested that the modulation of gut microbiota may be a viable strategy for the treatment of certain mental disorders [ 9].
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